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This pathway relies on an intracellular domain, TIRAF, which recruits adapter proteins such as MyD88, triggering NF-κβ-regulated genes and modulating inflammatory responses (Zheng et al., 2019

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In addition, it has been shown that 5-azacitidine (5-aza), a DNA hypomethylating agent, has synergistic effects when given with a topoisomerase 1 inhibitor, irinotecan, for the treatment of CRC [99], further complicating the role of DNA methylation in CRC