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Because catalase-dependent ethanol oxidation is driven by the availability of H 2 O 2 , its relative contribution is thought to increase under conditions of enhanced oxidative metabolism and high ethanol intake, thereby linking heavy drinking to intracerebral acetaldehyde production and region-specific neurotoxicity in the central nervous system [22,24]
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Key Takeaway While IV therapy may offer symptomatic relief and potential support for immune balance and recovery, the evidence remains inconclusive